Houston Texan ROY linebacker Brian Cushing, long thought the be a juicer at USC and in the NFL, and nailed with a positive PED test last spring is claiming 'overtrained athlete syndrome' affected his hormone status. Cushing also claimed that pigs fly and that the moon is made of green cheese.
Cushing tested positive of HCG, a hormone of pregnancy that supports testosterone when anabolic steroids are discontinued for say, PED testing. The NFL's Rook of the Year (yes we meant that) will sit out 4 games in 2010 in the steroid time out box.
There is a literature on Over-Training, which happens when a athlete stresses his body to a degree he cannot recover. The athlete's stress level is high; his HPA axis (hypothalmaic-pituitary-adrenal) can be messed up too. However, Brian Cushing needs to understand HCG isn't included in this deal...hormones like cortisone and testosterone may test somewhat abnormal.
Texan's owner Bob McNair will foolishly appeal to the NFL front office to rescind Cushing's suspension. While that indicates the owner supports his players, it also indicates Cushing is willing to maintain a fabrication while his owner humiliates himself.
Count us as very annoyed that a steroid-cheat like Cushing not only attains his career advancement by cheating, but continues to deceive caring people about his PED cheating. Cushing should fess up to the ch...there is only so much BS that is acceptable. Save others from the humiliation of defending your nefarious actions. That, or you were pregnant. (of course he can divulge all in his port-career book: Brain Cushing, Confessions of an Over-Trained and Dis-Ingenous Athlete)
Here is a legitimate abstract on the condition after the jump:
Sports Med. 1995 Oct;20(4):251-76. Blood hormones as markers of training stress and overtraining. Urhausen A, Gabriel H, Kindermann W. Institute of Sports and Preventive Medicine, University of Saarland, Saarbrücken, Germany.
An imbalance between the overall strain experienced during exercise training and the athlete's tolerance of such effort may induce overreaching or overtraining syndrome. Overtraining syndrome is characterised by diminished sport-specific physical performance, accelerated fatiguability and subjective symptoms of stress. Overtraining is feared by athletes yet there is a lack of objective parameters suitable for its diagnosis and prevention. In addition to the determination of substrates (e.g. lactate, ammonia and urea) and enzymes (e.g. creatine kinase), the possibilities for monitoring of training by measuring hormonal levels in blood are currently being investigated. Endogenous hormones are essential for physiological reactions and adaptations during physical work and influence the recovery phase after exercise by modulating anabolic and catabolic processes. Testosterone and cortisol are playing a significant role in metabolism of protein as well as carbohydrate metabolism. Both are competitive agonists at the receptor level of muscular cells. The testosterone/cortisol ratio is used as an indication of the anabolic/catabolic balance. This ratio decreases in relation to the intensity and duration of physical exercise, as well as during periods of intense training or repetitive competition, and can be reversed by regenerative measures. Correlations have been noted with the training-induced changes of strength. However, it seems more likely that the testosterone/cortisol ratio indicates the actual physiological strain in training, rather than overtraining syndrome. The sympatho-adrenergic system might be involved in the pathogenesis of overtraining. Overtraining appears as a disturbed autonomic regulation, which in its parasympathicotonic form shows a diminished maximal secretion of catecholamines, combined with an impaired full mobilisation of anaerobic lactic reserves. This is supposed to lead to decreased maximal blood lactate levels and maximal performance. Free plasma adrenaline (epinephrine) and noradrenaline (norepinephrine) may provide additional information for the monitoring of endurance training. While prolonged aerobic exercise conducted at intensities below the individual anaerobic threshold lead to a moderate rise of sympathetic activity, workloads exceeding this threshold are characterised by a disproportionate increase in the levels of catecholamines. In addition, psychological stress during competitive events is characterised by a higher catecholamines to lactate ratio in comparison with training exercise sessions. Thus, the frequency of training sessions with higher anaerobic lactic demands or of competition, should be carefully limited in order to prevent overtraining syndrome. In the state of overtraining syndrome and overreaching, respectively, an intraindividually decreased maximum rise of pituitary hormones (corticotrophin, growth hormone), cortisol and insulin has been found after a standardised exhaustive exercise test performed with an intensity of 10% above the individual anaerobic threshold.